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View Medical News Altered creatine kinase adenosine triphosphate kin...[Circulation. 2006] - PubMed Result [2/0] Vote Up Vote Down http://www.ncbi.nlm.nih.gov/sites/entrez?db=PubMed&cmd=Retri... Submitted by karuturi 15 months, 3 weeks, 5 days, 6 hours ago These first observations in human LVH demonstrate that it is not the relative or absolute CK metabolite pool sizes but rather the kinetics of ATP turnover through CK that distinguish failing from nonfailing hypertrophic hearts. Moreover, the deficit in ATP kinetics is similar in systolic and nonsystolic heart failure and is not related to the severity of hypertrophy but to the presence of CHF. Because CK temporally buffers ATP, these observations support the hypothesis that a deficit in myofibri Tags: creatine kinase comment (0) | Category: Cardiology | Add karuturi as a friend Comments Votes (my Friends) Votes (Everyone) Add your comment Add your comment If you already have an account, sign in: Email address: Password: If you don't, just complete these fields: (or join DoctorWorld.NET for free) Your name: Your site: (optional) Your comment: (You have up to 5000 characters left) Can you see the 5 most prominent letters/numbers in this image? (case insensitive) Please enter them here: « go back

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